S-benzylglutathione

Glutathione Analog / Tripeptide DerivativeRx: ResearchCompound: Research

Also known as: Glo1 inhibitor reference compound, S-Benzyl-GSH, S-Benzylglutathione tripeptide, γ-L-Glutamyl-S-benzyl-L-cysteinylglycine

Educational Only — Not medical advice. Consult a qualified clinician before using any peptide.

Summary

S-Benzylglutathione is a synthetic tripeptide (γ-Glu-Cys(S-Bn)-Gly) derived from glutathione by S-benzylation of the cysteine thiol. It is a well-characterized biochemical tool compound used to study the glyoxalase system and glutathione S-transferases. It has shown antiproliferative activity in cancer cell lines and is explored as a lead structure for antitumor and antiparasitic agents, but has no approved clinical use.

Mechanism of Action

Competitive inhibitor of glyoxalase I (Glo1); binds the active site of Glo1 by mimicking the hemithioacetal substrate, blocking conversion of methylglyoxal to S-lactoylglutathione and thereby disrupting detoxification of reactive carbonyl species. Also inhibits certain glutathione S-transferases.

Routes of Administration

In Vitro (cell Culture)Intraperitoneal (animal Studies Only)

Goals & Uses

  • Antiparasitic leadInfectious Disease ResearchLow
  • Anticancer activityOncologyLow
  • GST characterizationBiochemical ToolModerate
  • Glyoxalase I inhibitionEnzyme Inhibition / Biochemical ToolHigh

Contraindications

  • Human therapeutic administrationClinical UseHigh

Adverse Effects

  • CytotoxicityCell ViabilityUnknown

Drug Interactions

  • Methylglyoxal-metabolizing agentsLow

Population Constraints

  • General human populationRegulatory/safetyAbsolute

Regulatory Status

  • European UnionUnapprovedResearch reagent; no marketing authorization.
  • United StatesUnapprovedResearch chemical only; not an IND or approved drug.
  • United KingdomUnapprovedResearch use only; no MHRA authorization.

Not approved by any regulatory authority for human therapeutic use. Used exclusively as a research/laboratory reagent and pharmacological tool.

Evidence & Sources

No sources recorded yet.